Gap junctions mediate STAT5-independent beta-casein expression - by Antoine Abboud Khalil

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dc.contributor.authorKhalil, Antoine Abboud
dc.contributor.departmentDepartment of Biology
dc.contributor.facultyFaculty of Arts and Sciences
dc.contributor.institutionAmerican University of Beirut
dc.date2008
dc.date.accessioned2012-06-13T07:11:23Z
dc.date.available2012-06-13T07:11:23Z
dc.date.issued2008
dc.descriptionThesis (M.S.)--American University of Beirut, Dept. of Biology, 2008.;"Advisor : Dr. Rabih Talhouk, Professor , Biology --Co-Advisor : Dr. Marwan El Sabban, Professor ,Human Morphology --Member of Committee : Dr. Hala Gali-Muhtasib, Professor ,Biology --M
dc.descriptionBibliography : leaves 69-81.
dc.description.abstractBeta-casein gene expression has been shown to be regulated by prolactin signalin g and cell-ECM interaction, via the JAK2-STAT5 pathway. Few studies discuss the role of GJIC in Beta-casein expression. Our lab has previously established tha t enhanced GJIC
dc.format.extentxiii, 81 leaves : col. ill. 30 cm.
dc.identifier.urihttp://hdl.handle.net/10938/7697
dc.language.isoen
dc.relation.ispartofTheses, Dissertations, and Projects
dc.subject.classificationT:005110 AUBNO
dc.subject.lcshGap junctions (Cell biology)
dc.subject.lcshMammary glands
dc.subject.lcshEpithelial cells
dc.titleGap junctions mediate STAT5-independent beta-casein expression - by Antoine Abboud Khalil
dc.typeThesis

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