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Hypoalgesia and hyperalgesia induced by thymulin possible mediation by interleukin - 1[symbol] and prostaglandin - E2. - by Raffy Haroutin Jalakhian

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dc.contributor.author Jalakhian, Raffy Haroutin
dc.date.accessioned 2012-06-13T06:43:18Z
dc.date.available 2012-06-13T06:43:18Z
dc.date.issued 1996
dc.identifier.uri http://hdl.handle.net/10938/5223
dc.description Thesis (M.S.)--American University of Beirut. Department of Biology, 1996.;"Advisor: Dr. Salim Kanaan, Assistant Professor, Biology Co-Advisor: Dr. Bared Safieh-Garabedian, Assistant Professor, Biology -- Members of Committee: Dr. Nayef E. Saade, Profess
dc.description Bibliography: leaves 66-76.
dc.description.abstract There is considerable evidence for the existence of bi-directional irninune-neuro-endocrine interactions, representing an important homeostatic mechanism in the body. The aim of this study was to investigate the role of thymulin as a signalling molecule b
dc.format.extent xiii, 76 leaves : ill. cm.
dc.language.iso eng
dc.relation.ispartof Theses, Dissertations, and Projects
dc.subject.classification T:003704 AUBNO
dc.subject.lcsh Hyperalgesia.
dc.subject.lcsh Thymulin.
dc.subject.lcsh Interleukins.
dc.subject.lcsh Prostaglandins.
dc.title Hypoalgesia and hyperalgesia induced by thymulin possible mediation by interleukin - 1[symbol] and prostaglandin - E2. - by Raffy Haroutin Jalakhian
dc.title.alternative Hypoalgesia and hyperalgesia induced by thymulin
dc.type Thesis
dc.contributor.department American University of Beirut. Faculty of Arts and Sciences. Department of Biology


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