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Gap junctions mediate STAT5-independent beta-casein expression - by Antoine Abboud Khalil

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dc.contributor.author Khalil, Antoine Abboud
dc.date.accessioned 2012-06-13T07:11:23Z
dc.date.available 2012-06-13T07:11:23Z
dc.date.issued 2008
dc.identifier.uri http://hdl.handle.net/10938/7697
dc.description Thesis (M.S.)--American University of Beirut, Dept. of Biology, 2008.;"Advisor : Dr. Rabih Talhouk, Professor , Biology --Co-Advisor : Dr. Marwan El Sabban, Professor ,Human Morphology --Member of Committee : Dr. Hala Gali-Muhtasib, Professor ,Biology --M
dc.description Bibliography : leaves 69-81.
dc.description.abstract Beta-casein gene expression has been shown to be regulated by prolactin signalin g and cell-ECM interaction, via the JAK2-STAT5 pathway. Few studies discuss the role of GJIC in Beta-casein expression. Our lab has previously established tha t enhanced GJIC
dc.format.extent xiii, 81 leaves : col. ill. 30 cm.
dc.language.iso eng
dc.relation.ispartof Theses, Dissertations, and Projects
dc.subject.classification T:005110 AUBNO
dc.subject.lcsh Gap junctions (Cell biology)
dc.subject.lcsh Mammary glands
dc.subject.lcsh Epithelial cells
dc.title Gap junctions mediate STAT5-independent beta-casein expression - by Antoine Abboud Khalil
dc.type Thesis
dc.contributor.department American University of Beirut. Faculty of Arts and Sciences. Department of Biology


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